Pleuraldisease(胸膜疾病)ZhouLiuCancerHospitalofChineseAcademyofMedicalSciences,ShenzhenCenterPleuralDiseaseEpidemiology(流行病学):300/100,000eachyearBasicImagingFindingsPleuraleffusion(胸腔积液)Pleuralthickening(胸膜增厚)Masses(肿物)Pneumothrorax(气胸)ClassificationAsbestos-relatedbenignPleuralThickening(石棉相关的良性胸膜增厚)Non-asbestosrelatedbenignPleuraldisease(非石棉相关的良性胸膜疾病)MalignantPleuralthickening(恶性胸膜增厚)Pleuralfluid(includingEmpyema)胸腔积液(包括脓胸)Pneumothorax(气胸)RarePleuraltumors(罕见胸膜肿瘤)Fibroma(纤维瘤)Lipomasandliposarcomas(脂肪瘤和脂肪肉瘤)Imagingmodalities(成像方式)X-rayInitialchoiceUSIdentificationofpleuralfluid&pleuralnodularityGuidingpleuralprocedureHighlyoperatordependentPET-CTMalignantpleuraldiseaseCTPleuralthickeningPleuraleffusionPleuralmassesFattyattenuationorCalcificationMRISofttissueabnormalities(软组织)Youngerpatientsrequiringfollow-updelayof60-90sContrastpleuralphaseNormalappearance(正常表现)Fig.2.NormalCTwithpleural“intercostalstripe”(Arrow).Fig.1.Ultrasound:normallungwithpleuralstripe(WhiteArrow)and“comettail”artefacts伪影(outlinedbysmallwhitearrows).Asbestos-relatedbenignPleuralThickening(石棉相关的良性胸膜增厚)Fig.3.x-rayshowingdiffusethickening(arrow)andbluntingofcostophrenicangle(Right).Pleuralplaques(parietalinorigin)胸膜斑块(壁层胸膜起源)DiffusevisceralPleuralthickening弥漫性脏层胸膜增厚Asbestos-relatedbenignPleuralplaque(石棉相关良性胸膜斑块)Fig.4.A:CTimageshowingpleuralplaquesandassociatedinterstitiallines(“hairyplaques”)(Arrows).B:Roundedatelectasis球形肺不张(arrow)C:Pleurallesionsmimickingsuperimposednodularityinacasewithasbestos-relatedbenignpleuraldisease.Fig.6Increasedsubpleuralfattissueinacasewithasbestos-relatedbenignpleuraldiseaseFig.5Bilateralpleuralthickeninginacasewithasbestos-relatedbenignpleuraldisease.Non-asbestosrelatedbenignPleuraldisease(非石棉相关的良性胸膜疾病)DifferentCausesandsimilarimagingappearance(不同病因,相似影像表现)Priorempyema脓胸Paticularlytuberculosis结核ExtensivecalcificationVolumelossThickenedextra-PleuralfatlayerAssociatedparenchymalabnormalityPrevioustraumatichemothorax创伤性血胸PleuralcalcificationRibdeformityNormallungparenchymaPost-talcpleurodesis滑石粉胸腔固定术后ParietalpleuralthickeningHighattenuationtalcVisceralthickening“Sandwich”Infectious(感染性)Traumatic(创伤性)Post-procedure(手术操作后)TuberculouspleurisyTP结核性胸膜炎Fig.7CircumferentialsmoothpleuralthickeningandeffusionwithmediastinalpleuralinvolvementappearanceinaTPcase,pleuralthickeningislessthan1cm.Fig.8Irregularpleuralthickeningwithpleuralrindappearanceinapatientcausedbypleuraltuberculosis.Empyema(脓胸)Fig.9B.IncreasedsubpleuralfattissueinacasewithempyemaC.CTshowingempyemaand“splitpleura”sign(胸膜分离征)CFig.10Empyemawithpleuralsplitsign.50-year-oldwomanwithprogressivedyspneaandfever.CTshowscollectedleftpleuraleffusionswithpleuralsplitsign(arrow)(a,c)andpneumonicinfiltrate(b,d).TheMRIshowsonT2-weighted(T2W)imageshypointensebandsinthehyperintensepleuralfluid(e,f).Pneumoniaismorerestrictivethanthepleuraltransudateondiffusion-weightedimaging(DWI)(g,h).Post-talcpleurodesis滑石粉胸腔固定术后Fig.11A:CTpost-talconleftcircumferentialnodularpleuralthickeninginvolvingthemediastinalsurfacewithhighdensityelements(whitearrows).B:PETscandemonstratingactivitypost-talcpleurodesis(blackarrow).“Sandwich”MalignantPleuralthickening恶性胸膜增厚Metastaticdisease:majority&PrimaryPleuralmalignancy(Mesothelioma)Commonradiologicalappearance共性Irregularthickening>1cmNodularopacityPleuraleffusion:60%UsuallyunilateralChestwallinvasionandribdestructionMetastasisMesothelioma间皮瘤特性VolumelossCoexistedCalcifiedandnon-calcifiedPleuralplaquesInterstitialdiseaseorasbestosis:20%PriorasbestosexposureMalignantPleuralMesotheliomaMPM恶性胸膜间皮瘤Fig.12CircumferentialpleuralthickeningwithmediastinalpleuralinvolvementinMPMcases(pleuralrind).MesotheliomaInflammatoryPleuritisMetastasisFig.13ThreeCTimagesshowingfeaturesofmalignantdisease:nodularthickening(cross)andmediastinalinvolvement(arrow),bothinthepresenceofpleuraleffusion(E).MalignantPleuralThickening恶性胸膜增厚Fig.14(A)AsofttissuemassinaMPDcase.(B)Asoft-tissuemasswithcircumferentiallypleuralinvolvementinaMPMcase.MalignantPleuralThickening恶性胸膜增厚Fig.15.CTimage(A)andPET/CTimage(B)highlightingmalignantdisease(brightred).CautionFalsepositive:Infection(感染,如肺结核)Post-talcpleurodesis滑石粉胸腔固定术后Falsenegative:EpitheloidMesothelioma(上皮样间皮瘤)Pleuralfluid(includingEmpyema)胸腔积液(包括脓胸)Figa:Pleuraleffusion“meniscussign”Figb:LoculatedPleuraleffusionCauses:Empyema&HemothoraxAppearindifferentareassharpmedialmariginandhazylateralmarginOnlyevidentover200ml500mlmightappearnormalFig.16x-ray(A)pleuraleffusion(B)loculatedpleuraleffusionFig.17A:USshowingpleuraleffusion(E)withearlyseptations(arrows).“Infectedormalignanteffusions”B:USshowingorganisingpleuraleffusionwithheavymatureseptations(arrow).“honeycomb-likeappearance”Pleuralfluid(includingEmpyema)胸腔积液(包括脓胸)Pneumothorax(气胸)Fig.18.CXRandCTshowingpneumothorax.Arrowdemonstratingvisiblevisceralpleuraledge.Fibroma(纤维瘤)Fig.19A:CTshowinglargepleuralfibroma(*)withheterogeneouspatternpost-contrastB:MRI(STIRimage)showinglargepleuralfibroma(*).HomogeneousonunenhancedCTVaryinsizeCausingatelectasisinadjacentlungSmoothtaperingmarginObtuseangleatthejunctionofthemassandpleuraHeterogeneousaftercontrast(40%)Fig.20Solitaryfibroustumorwithnidussign.A40-year-oldwomanwithimpressionofelevatedrightdiaphagm(a,b).APET-CT(c,d,e)showsasupradiaphragmaticmass,withnoobviousFDGavidity(e).ThecoronalT2-weighted(T2W)withfatsaturationconfirmsthelesion(f),anddiffusionrestrictivepatternatb=0(g)andb=100suggeststhenidusbecauseofcentralsignalloss(arrowhead)(h).Thecentralnidusshowsthevascularstructure(arrowhead),hypointenseontheVIBEsequenceaftergadoliniumIVbecauseofsignalvoidphenomenon(arrow,iandj).Lipomasandliposarcomas(脂肪瘤和脂肪肉瘤)Fig.15.CTwithcontrastshowingpleurallipoma(*).A:Usinglungwindows;B:Usingmediastinalwindows(Notelowdensityoflipoma).Thankyou